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It modulates the immunosuppressive tumor microenvironment by regulating non-immune cells (such as cancer-associated fibroblasts, endothelial cells, and adipose stromal cells), repolarizing tumor-associated macrophages, activating NK cells, and tuning cytokines

BECN1 not only regulates autophagy but also affects cystine uptake by binding to System X c , leading to inhibition of GPX4 and increased levels of lipid peroxidation, inducing ferroptosis [64, 65]

Interestingly, Munne-Bosch et al

Semaglutide should be preferred as first-line AOM in people with obesity and established ASCVD or HFpEF, even in the absence of diabetes, based on robust CV benefit